Myocardial injury through serum troponin I and echocardiography in anaphylaxis: Takotsubo cardiomyopathy and the Kounis hypersensitivity–associated acute coronary syndrome

Nicholas G. Kounis; Andreas Mazarakis; Constantinos Bardousis

doi:10.1016/j.ajem.2015.12.019

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Myocardial injury through serum troponin I and echocardiography in anaphylaxis: Takotsubo cardiomyopathy and the Kounis hypersensitivity–associated acute coronary syndrome

Nicholas G. Kounis, MD, PhD

Department of Medical Sciences, Southwestern Greece Highest Institute of Education and Technology, Patras, Achaia, Greece

Andreas Mazarakis, MD, PhD

Department of Cardiology, “Saint Andrews” State General Hospital, Patras, Achaia, Greece

Constantinos Bardousis, MD

Department of Cardiology, “Saint Andrews” State General Hospital, Patras, Achaia, Greece

Department of Cardiology, University of Patras Medical Scholl, Rio, Patras, Achaia, Greece

DOI: http://dx.doi.org/10.1016/j.ajem.2015.12.019

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Measuring of high-sensitivity cardiac troponin levels is essential in the diagnosis of acute myocardial infarction. Acute myocardial infarction is defined as myocardial cell death due to prolonged myocardial ischemia. Indeed, in the third universal definition of myocardial infarction [1], detection of rise and/or fall of cardiac troponin above the 99th percentile of the upper reference limit together with ischemic symptoms, electrocardiographic changes of new ischemia, development of pathologic Q-waves in the electrocardiogram, or imaging evidence of new loss of viable myocardium or new regional wall motion abnormality is essential for defining myocardial infarction.

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Myocardial injury through serum troponin I and echocardiography in anaphylaxis: Takotsubo cardiomyopathy and the Kounis hypersensitivity–associated acute coronary syndrome

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